• Intro
• Preface
• Contents
• Contributors
• Part I: Nausea and Vomiting
• Chapter 1: Approach to Nausea and Vomiting
• Case Study
• Objectives
• Epidemiology
• Pathophysiology
• Etiology
• Diagnostic Evaluation
• Treatment
• Clinical Case Study Follow-Up
• Self-Test
• References
• Essential Reading
• Chapter 2: Gastroparesis
• Case Study
• Objectives
• Epidemiology
• Symptoms
• Diagnostic Evaluation
• Treatment
• Symptom Management
• Prokinetics
• Prokinetics in Development
• Pyloric Interventions
• Gastric Electrical Stimulation
• Case Study: Follow-Up
• Self-Test
• References
• Essential Reading
• Chapter 3: Nausea and Vomiting of Pregnancy and Postoperative Nausea and Vomiting
• Case Study 1
• Case Study 2
• Objectives
• Nausea and Vomiting of Pregnancy
• Epidemiology
• Etiology/Pathophysiology
• Diagnosis
• Treatment
• First-Line Therapy
• Second-Line Treatment
• Third-Line Treatment
• Postoperative Nausea and Vomiting
• Epidemiology
• Pathophysiology
• Etiology
• Diagnosis
• Treatment
• 5-Hydroxytryptamine Receptor Antagonists
• Corticosteroids
• Anticholinergics
• Neurokinin-1 Receptor Antagonists
• Dopamine Receptor Antagonists
• Antihistamines
• Other PONV Treatment Options
• Case Study: Follow-Up
• Self-Test Questions
• References
• Essential Reading List
• Chapter 4: Cyclic Vomiting Syndrome and Cannabinoid Hyperemesis Syndrome
• Case Study
• Objectives
• Epidemiology
• Etiology/Pathophysiology
• Symptoms
• Diagnostic Evaluation
• Treatment
• Case Study: Follow-Up
• Self-Test
• References
• Essential Reading
• Chapter 5: Chronic Intestinal Pseudo-Obstruction
• Case Study
• Objectives
• Epidemiology
• Classification
• Etiology and Pathophysiology
• Neuropathies
• Intestinal Myopathies
• Mesenchymopathies
• Symptoms
• Diagnosis
• Treatment
• Nutrition Assessment
• Oral Diet
• Enteral Nutrition Considerations
• Parenteral Nutrition Considerations
• Medications and Other Therapies
• Fecal Transplant
• GI Decompression
• Small Intestinal Transplantation
• Case Study Follow-Up
• Self-Test
• References
• Essential Reading
• Part II: Dyspepsia and Other Disorders of Neuromuscular Function
• Chapter 6: A Diagnostic Approach to Dyspepsia
• Case Study
• Objectives
• Epidemiology
• Etiology and Pathophysiology
• Symptoms
• Diagnostic Evaluation
• Treatment
• Case Study: Follow-Up
• Self-Test
• References
• Essential Reading
• Chapter 7: Helicobacter pylori and Related Diseases
• Case
• Objectives
• Epidemiology
• Etiology and Pathophysiology
• Symptoms
• Diagnostic Evaluation
• Treatment
• Case Study: Follow-Up
• Self-Test
• References
• Essential Reading
• Chapter 8: Functional Dyspepsia
• Case
• Objectives
• Epidemiology
• Etiology and Pathophysiology
• Symptoms
• Diagnostic Evaluation
• Treatment
• Helicobacter Pylori
• Acid Suppression
• Antidepressants
• Prokinetics
• Further Options
• Case Study: Follow-Up
• Self-Test
• References
• Essential Reading
• Chapter 9: Rumination Syndrome
• Case Study
• Objectives
• Epidemiology
• Etiology and Pathophysiology
• Symptoms
• Diagnostic Evaluation
• Treatment
• Case Study: Follow-Up

This casebook provides a concise yet comprehensive state-of-the art review of common stomach and small intestine disorders. The casebook is divided into five parts, each of which focuses on a major disorder, symptom, or clinical scenario related to the stomach and small intestine, including dyspepsia, small bowel disorders, nausea and vomiting, chronic abdominal pain, and post-operative GI surgery challenges. Each part is comprised of cases illustrating different aspects of the subject, with each case containing sections on case presentation, objectives, epidemiology and etiology, diagnostic evaluation and treatment, case follow-up, clinical pearls, and Q&A. Some major topics presented in these cases include nausea and vomiting in the pregnant patient, gas-bloat, functional abdominal pain, post-GI surgery complications, and celiac disease and non-celiac gluten sensitivity. Written by internationally renowned experts in the field, Essential Medical Disorders of the Stomach and Small Intestine: A Clinical Casebook is a valuable resource for clinicians and practitioners who treat patients afflicted with disorders of the stomach and small intestine.

• Intestinal failure in children
• Short Bowel Syndrome-Intestinal Adaptation
• Bacterial Overgrowth and Intestinal Microbiome
• Intestinal failure-associated liver disease
• Recent Advances in Nutritional Care of Patients with Intestinal Failure
• Pharmacological improvement of bowel function and adaptation
• Modern Intestinal rehabilitation and outcomes
• Serial transverse enteroplasty
• Longitudinal intestinal lengthening
• Controlled tissue expansion
• Pediatric small bowel transplantation
• an update
• Intestinal Regeneration
• distraction enterogenesis.

This book provides readers with a succinct but comprehensive overview of current concepts relating to intestinal failure, including its treatment, with a particular focus on recent advancements. The opening chapters address basic aspects of intestinal failure, including etiology, clinical pathophysiology, physiology of intestinal adaptation, and intestinal microbiota. The complications of intestinal failure, such as bacterial overgrowth and intestinal failure-associated liver disease, are then discussed. A series of chapters specifically address important strategies in the nutritional, pharmacological, and surgical treatment of intestinal failure as well as the concept of intestinal rehabilitation. Finally, two significant emerging developments are considered: regenerative medicine and distraction enterogenesis. The authors are internationally recognized experts from Europe and North America who are ideally placed to describe current knowledge and progress in the field.

• 1. Immunosuppression in solid organ transplantation
• 2. Infection Complications after Abdominal Organ Transplantation
• 3. National Transplant Network. Donor selection and evaluation criteria
• 4. The safety of donor
• 5. Medical disease after abdominal solid organ transplantation and the risk of solid and hematologic malignant tumor after transplantation)
• 6 Indication and timing
• 7. The donor procurement
• 8. Orthotopic liver transplantation
• 9. Anesthetic and perioperative management for liver transplantation
• 10 Living donor liver transplantation
• 11. Hepatic Acute and chronic rejection
• 12. Postoperative technical complications
• 13. Percutaneous management of biliary and vascular complications
• 14. Re-transplantation (Causes, Outcome)
• 15. Indication and timing
• 16. The donor procurement: Standard technique for procurement in cadaveric donors
• 17. Live donor evaluation
• 18. Living donor nephrectomy
• 19. Renal Transplantation: Surgical technique
• 20. The intraoperative and perioperative management of Kidney transplantation
• 21. Kidney acute and chronic rejection
• 22. Kidney Transplantation: Post-Operative Technical Complications
• 23. Indications and timing
• 24. The donor procurement
• 25 Transplantation
• 26 Abdominal wall transplantation and technique
• 27. Anesthetic and perioperative management for Intestinal transplantation
• 28. Segmental Living-related Small Bowel Transplantation
• 29. Intestinal Acute and Chronic rejection
• 30. Combined Kidney-Liver Transplantation
• 31. Kidney-pancreas Transplantation.

• Aloe vera: a Natural Healing Agent for Gastro-intestinal and Chronic Inflammatory Conditions
• Chondroitin Sulphate and Glucosamine Sulphate as Potential Disease-Modifying Agents for Osteoarthritis
• Probiotics as Natural Healing Agents for Gastro-intestinal Diseases and Arthritis.- Therapeutic Effects of Ribonucleinate (Ribonucleotides) in Immuno-inflammatory and Arthritic Diseases
• The Medicinal Properties and Phytochemistry of Plants of the Genus Terminalia (Combretaceae)
• Celery Seed and Related Extracts with Anti-athritic, Anti-Ulcer and anti-Microbial Activities
• Lyprinol, Lipids and other Components of Green Lipped Mussel as Anti-Arthritic and Anti-Ulcer Agents
• Zinc Compounds as Topical and Oral Anti-Arthritic and Anti-ulcer Compounds.

The new series A-Z Notes in Radiological Practice and Reporting provides practical guides for residents and general radiologists, organized alphabetically, primarily according to disease or condition. All booklets are designed so as to cover a large spectrum of topics referring to different anatomical regions of interest. Entries typically include a short description of pathological and clinical characteristics, guidance on selection of the most appropriate imaging technique, a schematic review of potential diagnostic clues, and useful tips and tricks. The present booklet, enriched by illustrations and schemes, is devoted to gastrointestinal imaging. Major topics in CT and MR imaging of the small bowel, colon, rectum, and anus are treated concisely in alphabetical order. For each topic a brief review of clinical features and pathology is presented, followed by a short description of imaging technique and an accurate review of imaging findings and signs which are useful in the differential diagnosis of gastrointestinal disease.

• Part 1. Basic concepts underlying the microbiota-gut-brain axis. Microbial endocrinology and the microbiota-gut-brain axis / Mark Lyte
• Utilizing "omics" tools to study the complex gut ecosystem / Anthony Fodor
• The enteric nervous system and gastrointestinal innervation: integrated local and central control / John B. Furness, Brid P. Callaghan, Leni R. Rivera, and Hyun-Jung Cho
• Intestinal barrier function and the brain-gut axis / Carmen Alonso, María Vicario, Marc Pigrau, Beatriz Lobo, and Javier Santos
• Vagal pathways for microbiome-brain-gut axis communication / Paul Forsythe, John Bienenstock, and Wolfgang A. Kunze
• The brain-gut axis in health and disease / Yasser Al Omran and Qasim Aziz
• part 2. Mechanistic factors influencing the microbiota-gut-brain axis. Gastrointestinal hormones and their targets / Jens F. Rehfeld
• Microbiome, HPA axis and production of endocrine hormones in the gut / Nobuyuki Sudo
• Neuropeptides and the microbiota-gut-brain axis / Peter Holzer and Aitak Farzi
• Bacterial neuroactive compounds produced by psychobiotics / Rebecca Wall, John F. Cryan, R. Paul Ross, Gerald F. Fitzgerald, Timothy G. Dinand, and Catherine Stanton
• Multidirectional chemical signalling between mammalian hosts, resident microbiota, and invasive pathogens: neuroendocrine hormone-induced changes in bacterial gene expression / Michail H. Karavolos and C.M. Anjam Khan
• Influence of stressor-induced nervous system activation on the intestinal microbiota and the importance for immunomodulation / Michael T. Bailey
• part 3. The microbiota-gut-brain axis in health and disease. The effects of inflammation, infection and antibiotics on the microbiota-gut-brain axis / Premysl Bercik and Stephen M. Collins
• Microbiota, inflammation and obesity / Yolanda Sanz and Angela Moya-Pérez
• Microbiota, immunoregulatory old friends and psychiatric disorders / Graham A.W. Rock, Charles L. Raison, and Christopher A. Lowry
• Microbiota-gut-brain axis and cognitive function / Mélanie G. Gareau
• The impact of microbiota on brain and behavior: mechanisms & therapeutic potential / Yuliya E. Borre, Rachel D. Moloney, Gerard Clarke, Timothy G. Dinan, and John F. Cryan
• Neuroimaging the microbiome-gut-brain axis / Kirsten Tillisch and Jennifer S. Labus
• The future of probiotics for disorders of the brain-gut axis / Eamonn M.M. Quigley and Fergus Shanahan
• Index.

"The field of microbial endocrinology is expressly devoted to understanding the mechanisms by which the microbiota (bacteria within the microbiome) interact with the host ("us"). This interaction is a two-way street and the driving force that governs these interactions are the neuroendocrine products of both the host and the microbiota. Chapters include neuroendocrine hormone-induced changes in gene expression and microbial endocrinology and probiotics. This is the first in a series of books dedicated to understanding how bi-directional communication between host and bacteria represents the cutting edge of translational medical research, and hopefully identifies new ways to understand the mechanisms that determine health and disease"--Publisher's description.

• A review of the epidemiology of inflammatory bowel disease with a focus on diet, infections, and antibiotic exposure / Lewis, J.D.
• Current issues in the design of clinical trials in IBD / Feagan, B.G.
• The intestinal microbiota in inflammatory bowel diseases / Sartor, R.B.
• Genetics and innate and adaptive immunity in IBD / Zanello, G.; Kevans, D.; Goethel, A.; Silverberg, M.; Tyler, A.; Croitoru, K.
• Interplay of nutrients and microbial metabolites in intestinal immune homeostasis : differential and common mechanisms of immune regulation in the small bowel and colon / Perrigoue, J.; Das, A.; Mora, J.R.
• Diet, the gut microbiome, and the metabolome in IBD / Wu, G.D.
• Antibiotics, probiotics, and prebiotics in IBD / Bernstein, C.N.
• Fecal microbiota transplantation : an interest in IBD / Kahn, S.A.; Goeppinger, S.R.; Rubin, D.T.
• Enteral nutrition as treatment option for Crohn's disease : in kids only / Ruemmele, F.M.; Pigneur, B.; Garnier-Lenglin, H.
• Effects of exclusive enteral nutrition on bone mass, linear growth, and body composition in children with Crohn's disease / Sylvester, F.A.
• Exclusive enteral nutrition : clues to the pathogenesis of Crohn's disease / Levine, A.
• Current state of the art of medical foods / Blum, S. ; Brito, F.
• IBD therapy : new targets and unmet needs / Colombel, J.-F.

Toxoplasma gondii is an obligate intracellular parasite. It has extraordinary abilities to infect virtually any warm blooded animal, cross a number of biological barriers (intestine, placenta and blood-brain), and invade any nucleated cell tested thus far in vitro. Chapter 1 provides a general introduction to Toxoplasma gondii and its ability to invade the host cell. Emphasis is placed on biological barriers and molecular mechanism of invasion for the different forms of the parasite. Chapter 2 describes a study of how Toxoplasma crosses the human placenta. Chapter 3 provides an insight as to how Enteric Glial Cells contribute to oral Toxoplasma infection in vivo. Chapter 4 deals with the study of how Toxoplasma sporozoites invade host cells. All research topics shed light on details of how Toxoplasma crosses the anatomical barriers: from the tissue barriers of placenta to infection to the possible immune and barrier functions of Enteric Glial Cells to how Toxoplasma sporozoites invade host cells. Chapter 2 describes experiments demonstrating that Toxoplasma tachyzoites invade the human placenta. These experiments used the human placental explant system to determine where the tachyzoites first invade, how they travel down the anchoring villi, and whether there exist type differences in ability to invade this tissue. This study provided several insights: (i) Toxoplasma tachyzoites are unable to penetrate the syncytium, and invade only the exposed tissue (anchoring villi); (ii) There does not appear to be a significant difference in the ability of different strains to infect the tissue. Chapter 3 addresses the possible role of TGF-[beta] by Enteric Glial Cells (EGCs) in oral Toxoplasma infection. Mice expressing a Dominant Negative TGF-[beta] receptor in EGCs were orally infected with Toxoplasma tissue cysts. At the end of the time trials, mice were sacrificed and their intestines subjected to histological analysis. Experiments did not reveal a clear-cut contribution to the control of inflammation by this cell type to the progress of disease. Chapter 4 examines the function of two paralogues of AMA1 and RON2 that are present exclusively in Toxoplasma sporozoites. Dubbed sporoAMA1 and sporoRON2, respectively, that sporoRON2 and sporoAMA1 form an exclusive pairing, stabilized with unique structural features. Furthermore, pre-treating sporozoites with a C-terminal portion of sporoRON2 (but not generic RON2) inhibits sporozoite invasion. We see sporoRON2 in a different compartment from RON4, suggesting that sporoRON2 functions independently of known moving junction components. These data indicate that sporozoites' host cell invasion is dependent on a novel, stage-specific version of the AMA1-RON2 pairing.

Overview: 555 million preschool children live in developing countries where malnutrition (undernutrition) is a significant problem. The factors that contribute to this issue are multiple, with two important factors being gut microbiota and gut function. To begin to understand gut microbiota and gut function in a developing country, we performed two parallel studies: in the first study, we characterized the baseline gut microbiota of healthy Bangladeshi children, and in the second study, we investigated the association between household environmental conditions and gut function in Bangladeshi children. The gut microbiota affect nutrient metabolism, immune function, and pathogen resistance. Loss of gut function as occurs in environmental enteropathy (a multifaceted, subclinical intestinal disorder likely derived from repeated episodes of infectious gastroenteritis and chronic inflammation) contributes to malnutrition and growth faltering. It is possible that these processes are interrelated with the gut microbiota serving a critical role in environmental enteropathy. Gut microbiota: Previous studies have focused on the gut microbiota of infants and adults from developed countries, but few have characterized the gut microbiota of people living in developing countries. Furthermore, little is known about the gut microbiota and its variation over time in primary-school-age children and adolescents in either developed or developing countries. In this study, we compared the monthly variation in fecal microbiota composition of healthy children (ages 8--14 years) living in an urban slum in Bangladesh with that of age-matched children from affluent suburban communities in the United States. We analyzed 8,000 near full-length sequences of the small-subunit ribosomal RNA gene (16S rDNA) and over 845,000 pyrosequencing reads spanning the V1-V3 variable region of the 16S rDNA. The distal gut of Bangladeshi children harbored significantly higher microbial diversity than U.S. children, a result validated by the discovery of several novel lineages from various bacterial phyla in the full-length Bangladeshi dataset. Bangladeshi and U.S. children displayed distinct gut microbiota architectures. Moreover, significantly lower month-to-month temporal stability was observed in Bangladeshi children compared to U.S. children. Together, these results suggest that different environmental or genetic variables may affect the microbiota of healthy children in the two countries. Further studies are necessary to investigate the underlying mechanisms of these differences and to incorporate these insights into prevention or treatment of diseases. Gut function: Although poor water quality, sanitation, and hygiene are putative risk factors for environmental enteropathy, there are no studies investigating the relationship of household environmental conditions to environmental enteropathy. We compared levels of environmental enteropathy markers to growth patterns and measured the prevalence of parasitic infections in children living in different levels of environmental cleanliness based on indictors of water, sanitation, and hygiene in their households. We conducted a follow-up study of 119 children (< 4 years old) from an existing cohort, living in rural Bangladesh and divided into two types of households: a 'mitigated household' had good water quality, improved sanitation, and a handwashing station stocked with water and soap, and a 'contaminated household' had poor water quality, and inadequate sanitation and handwashing facilities. We measured anthropometry, intestinal parasitic infection, and the following markers to assess gut function: Lactulose:Mannitol (L:M) ratio, immunoglobulin G endotoxin core antibody (IgG EndoCAb), and total IgG. After adjustment for potential confounding, children from mitigated households had higher height-for-age Z-scores (HAZ), lower L:M ratios, and lower IgG EndoCAb titers than children from contaminated households. The L:M ratio was also strongly associated with HAZ in the cohort. Children living in environmentally mitigated households had lower levels of parasitic infection, improved measures of gut function, and improved growth compared to children living in contaminated environments. These results support the hypothesis that environmental contamination mediated through environmental enteropathy could be a cause of growth faltering in low-income countries. Interpretation: By characterizing the baseline gut microbiota of healthy children living in a developing country and correlating their gut function with household environmental conditions, our work provides a foundation to address the role of the gut microbiota and environmental enteropathy in child health.

The intestinal epithelium is one of the most rapidly proliferating tissues in the body. A complete turnover of the epithelium occurs every 3-5 days in the mouse, a process that is maintained by a small population of intestinal stem cells (ISCs) that reside in the crypt bases. The signals that regulate the behavior of these ISCs are still poorly understood. However, the recent identification of genes that mark functional stem cells has yielded insights into how ISCs are regulated and maintained. Here, we demonstrate that Bmi1 and Lgr5 mark two functionally distinct ISCs in vivo. Lgr5 marks mitotically active ISCs that exhibit exquisite sensitivity to canonical Wnt modulation, contribute robustly to homeostatic regeneration, and are quantitatively ablated by irradiation. In contrast, Bmi1 marks quiescent ISCs that are insensitive to Wnt perturbations, contribute weakly to homeostatic regeneration, and are resistant to high dose radiation injury. Post-irradiation, however, the normally quiescent Bmi1+ ISCs dramatically proliferate to clonally repopulate multiple contiguous crypts and villi. Clonogenic culture of isolated single Bmi1+ ISCs yields long-lived self-renewing spheroids of intestinal epithelium that produce Lgr5-expressing cells, thereby establishing a lineage relationship between these two populations in vitro. Taken together, these data provide direct evidence that Bmi1 marks quiescent, injury-inducible reserve ISCs that exhibit striking functional distinctions from Lgr5+ ISCs, and support a model whereby distinct ISC populations facilitate homeostatic versus injury-induced regeneration. How these ISCs maintain their stemness remains unclear. Paneth cells have been suggested to serve as niche cells for the Lgr5+ ISCs, perhaps through the secretion of essential paracrine factors, but recent reports clearly demonstrate that Paneth cells are not required for Lgr5+ ISC maintenance. Recently, the G-protein-coupled receptors Lgr4-6 were reported to associate with Wnt receptors to mediate R-spondin signaling. Given the importance of Wnt/R-spondin signaling in intestinal crypt maintenance, we tested the in vivo function of Lgr5 by adenovirus-mediated overexpression of the soluble ligand-binding Lgr5 extracellular domain. Circulating Lgr5 ectodomain induced the migration of Paneth cells from the crypts, their eventual loss, and a concomitant disappearance of Lgr5+ ISCs. Paneth cell migration was associated with downregulation of Wnt signaling and its target EphB3. The loss of Lgr5+ ISCs did not affect maintenance of the intestinal epithelium, nor did Lgr5+ stem cells disappear from non-intestinal organs. Together, these findings characterize an easily tractable experimental model for the in vivo deletion of Lgr5+ ISCs, and suggest that Lgr receptors function to actively maintain Lgr5+ ISCs in vivo.

The importance of the intestinal microbiota to the health of an organism is well recognized. Alterations in the composition of the microbiota are associated with a number of diseases. However, the extent to which the microbiota varies across healthy individuals and time and the causes of this variation are still not fully understood. In order to distinguish between microbial community states of health and disease, the range of healthy microbial communities must be determined. In addition, the effect of perturbations on the microbiota is an area of active research. Recent studies indicate that intestinal infections can have a significant impact on the composition of intestinal microbiota. However, the extent and duration of these changes during different infections have not been fully characterized. My aims were to characterize the range of healthy microbiota in an inbred mouse population over time and to determine the dynamics of alterations in the microbiota during disease. I also wanted to determine if certain characteristics of the microbiota make animals more or less susceptible to infection. In this work, 46 individual healthy mice were sampled over time to assess the range of microbial communities in health. A mouse model of Salmonella enterica serovar Typhimurium infection was used to assess the impact of infection on the microbiota. Mice were sampled prior to infection and over the course of infection. This work finds that the microbiota of healthy mice varies significantly over time and among individuals. However, there are stable differences among individual mice, which may be established at the point of weaning when adult microbial communities develop. In addition, this work finds significant changes in the microbiota during infection that differ with the level of infection. This data also shows that there are some taxa whose abundance prior to infection correlates with outcome of infection. This work provides insights into the dynamics of the microbiota in health and disease and its impact on the health of the host.

• Functional morphology of the gastrointestinal tract / L. Shen
• Vibrio cholerae interactions with the gastrointestinal tract: Lessons from animal studies / J.M. Ritchie and M.K. Waldor
• Interaction of Yersinia with the gut: Mechanisms of pathogenesis and immune evasion / P. Dube
• Molecular mechanisms of Salmonella virulence and host resistance / Y. Valdez, R.B.R. Ferreira and B.B. Finlay
• Virulence mechanisms and persistence strategies of the human gastric pathogen Helicobacter pylori / tW. Fischer, S. Prassl and R. Haas
• Listeria as an enteroinvasive gastrointestinal pathogen / S.B. Barbuddhe and T. Chakraborty
• Molecular mechanisms of Campylobacter infection / J.P.M. Putten ... [et al.]
• Shigella infection of intestinal epithelium and circumvention of the host innate defense system / H. Ashida ... [et al.].

• Role of innate immune system and host-commensal mutualism / S. Rakoff-Nahoum and R. Medzhitov
• Molecular networks orchestrating GALT development / D. Finke and D. Meier
• Lymphoid tissue inducer cells in intestinal immunity / I.I. Ivanov, G.E. Diehl, and D.R. Littman
• Specificity and plasticity of memory lymphocyte migration / J. Rodrigo Mora and U.H. von Adrian
• IgA adaptation to the presence of commensal bacteria in the intestine / A. J. Macpherson
• Intestinal IgA synthesis : a primitive form of adaptive immunity that regulates microbial communities in the gut / S. Fagarasan
• B cell recruitment and selection in mouse GALT germinal centers / S. Casola and K. Rajewsky
• Structural insights into antibody-mediated mucosal immunity / A.E. Hamburger, P.J. Bjorkman, and A.B. Herr.